80 FR 20189 - Addition of 1-Bromopropane; Community Right-To-Know Toxic Chemical Release Reporting
ENVIRONMENTAL PROTECTION AGENCY Federal Register Volume 80, Issue 72 (April 15, 2015)
Federal Register Volume 80, Issue 72 (April 15, 2015)
| Page Range | 20189-20195 | |
| FR Document | 2015-08664 |
[Federal Register Volume 80, Number 72 (Wednesday, April 15, 2015)] [Proposed Rules] [Pages 20189-20195] From the Federal Register Online [www.thefederalregister.org] [FR Doc No: 2015-08664] ======================================================================= ----------------------------------------------------------------------- ENVIRONMENTAL PROTECTION AGENCY 40 CFR Part 372 [EPA-HQ-TRI-2015-0011; FRL-9925-29-OEI] RIN 2025-AA41 Addition of 1-Bromopropane; Community Right-To-Know Toxic Chemical Release Reporting AGENCY: Environmental Protection Agency (EPA). ACTION: Proposed rule. ----------------------------------------------------------------------- SUMMARY: The Environmental Protection Agency (EPA) is proposing to add 1-bromopropane to the list of toxic chemicals subject to reporting under section 313 of the Emergency Planning and Community Right-to-Know Act (EPCRA) of 1986 and section 6607 of the Pollution Prevention Act (PPA) of 1990. 1-Bromopropane has been classified by the National Toxicology Program in their 13th Report on Carcinogens as ``reasonably anticipated to be a human carcinogen.'' EPA believes that 1- bromopropane meets the EPCRA section [[Page 20190]] 313(d)(2)(B) criteria because it can reasonably be anticipated to cause cancer in humans. Based on a review of the available production and use information, 1-bromopropane is expected to be manufactured, processed, or otherwise used in quantities that would exceed the EPCRA section 313 reporting thresholds. DATES: Comments must be received on or before June 15, 2015. ADDRESSES: Submit your comments, identified by Docket ID No. EPA-HQ- TRI-2015-0011, by one of the following methods:www.regulations.gov: Follow the on-line instructions for submitting comments. Email: oei.docket@epa.gov. Mail: Office of Environmental Information (OEI) Docket, Environmental Protection Agency, Mail Code: 28221T, 1200 Pennsylvania Ave. NW., Washington, DC 20460. Hand Delivery: EPA Docket Center (EPA/DC), EPA West, Room 3334, 1301 Constitution Ave. NW., Washington, DC 20460. Such deliveries are only accepted during the Docket's normal hours of operation, and special arrangements should be made for deliveries of boxed information. Instructions: Direct your comments to Docket ID No. EPA-HQ-TRI- 2015-0011. EPA's policy is that all comments received will be included in the public docket without change and may be made available online at www.regulations.gov, including any personal information provided, unless the comment includes information claimed to be Confidential Business Information (CBI) or other information whose disclosure is restricted by statute. Do not submit information that you consider to be CBI or otherwise protected through www.regulations.gov or email. The www.regulations.gov Web site is an ``anonymous access'' system, which means EPA will not know your identity or contact information unless you provide it in the body of your comment. If you send an email comment directly to EPA without going through www.regulations.gov, your email address will be automatically captured and included as part of the comment that is placed in the public docket and made available on the Internet. If you submit an electronic comment, EPA recommends that you include your name and other contact information in the body of your comment and with any disk or CD-ROM you submit. If EPA cannot read your comment due to technical difficulties and cannot contact you for clarification, EPA may not be able to consider your comment. Electronic files should avoid the use of special characters, avoid any form of encryption, and be free of any defects or viruses. Docket: All documents in the docket are listed in the www.regulations.gov index. Although listed in the index, some information is not publicly available, e.g., CBI or other information whose disclosure is restricted by statute. Certain other material, such as copyrighted material, will be publicly available only in hard copy. Publicly available docket materials are available either electronically in www.regulations.gov or in hard copy at the OEI Docket, EPA/DC, EPA West, Room 3334, 1301 Constitution Ave. NW., Washington, DC. This Docket Facility is open from 8:30 a.m. to 4:30 p.m., Monday through Friday, excluding legal holidays. The telephone number for the Public Reading Room is (202) 566-1744, and the telephone number for the OEI Docket is (202) 566-1752. FOR FURTHER INFORMATION CONTACT: Daniel R. Bushman, Environmental Analysis Division, Office of Information Analysis and Access (2842T), Environmental Protection Agency, 1200 Pennsylvania Ave. NW., Washington, DC 20460; telephone number: 202-566-0743; fax number: 202- 566-0677; email: bushman.daniel@epa.gov, for specific information on this notice. For general information on EPCRA section 313, contact the Emergency Planning and Community Right-to-Know Hotline, toll free at (800) 424-9346 (select menu option 3) or (703) 412-9810 in Virginia and Alaska or toll free, TDD (800) 553-7672, http://www.epa.gov/superfund/contacts/infocenter/. SUPPLEMENTARY INFORMATION: I. General Information A. Does this notice apply to me? You may be potentially affected by this action if you manufacture, process, or otherwise use 1-bromopropane. Potentially affected categories and entities may include, but are not limited to: ------------------------------------------------------------------------ Examples of potentially Category affected entities ------------------------------------------------------------------------ Industry............................... Facilities included in the following NAICS manufacturing codes (corresponding to SIC codes 20 through 39): 311 *, 312 *, 313 *, 314 *, 315 *, 316, 321, 322, 323 *, 324, 325 *, 326 *, 327, 331, 332, 333, 334 *, 335 *, 336, 337 *, 339 *, 111998 *, 211112 *, 212324 *, 212325 *, 212393 *, 212399 *, 488390 *, 511110, 511120, 511130, 511140 *, 511191, 511199, 512220, 512230 *, 519130 *, 541712 *, or 811490 *. * Exceptions and/or limitations exist for these NAICS codes. Facilities included in the following NAICS codes (corresponding to SIC codes other than SIC codes 20 through 39): 212111, 212112, 212113 (correspond to SIC 12, Coal Mining (except 1241)); or 212221, 212222, 212231, 212234, 212299 (correspond to SIC 10, Metal Mining (except 1011, 1081, and 1094)); or 221111, 221112, 221113, 221119, 221121, 221122, 221330 (Limited to facilities that combust coal and/or oil for the purpose of generating power for distribution in commerce) (corresponds to SIC 4911, 4931, and 4939, Electric Utilities); or 424690, 425110, 425120 (Limited to facilities previously classified in SIC 5169, Chemicals and Allied Products, Not Elsewhere Classified); or 424710 (corresponds to SIC 5171, Petroleum Bulk Terminals and Plants); or 562112 (Limited to facilities primarily engaged in solvent recovery services on a contract or fee basis (previously classified under SIC 7389, Business Services, NEC)); or 562211, 562212, 562213, 562219, 562920 (Limited to facilities regulated under the Resource Conservation and Recovery Act, subtitle C, 42 U.S.C. 6921 et seq.) (corresponds to SIC 4953, Refuse Systems). Federal Government..................... Federal facilities. ------------------------------------------------------------------------ This table is not intended to be exhaustive, but rather provides a guide for readers regarding entities likely to be affected by this action. Some of the entities listed in the table have exemptions and/or limitations regarding coverage, and other types of entities not listed in the table could also be affected. To determine whether your facility would be affected by this action, you should carefully examine the applicability criteria in part 372 subpart B of Title 40 of the Code of Federal Regulations. If you have questions regarding the applicability of this action to a particular entity, consult the person listed in the preceding FOR FURTHER INFORMATION CONTACT section. [[Page 20191]] II. Introduction A. What is the statutory authority for this proposed rule? This rule is issued under EPCRA section 313(d) and section 328, 42 U.S.C. 11023 et seq. EPCRA is also referred to as Title III of the Superfund Amendments and Reauthorization Act of 1986. B. What is the background for this action? Section 313 of EPCRA, 42 U.S.C. 11023, requires certain facilities that manufacture, process, or otherwise use listed toxic chemicals in amounts above reporting threshold levels to report their environmental releases and other waste management quantities of such chemicals annually. These facilities must also report pollution prevention and recycling data for such chemicals, pursuant to section 6607 of the PPA, 42 U.S.C. 13106. Congress established an initial list of toxic chemicals that comprised 308 individually listed chemicals and 20 chemical categories. EPCRA section 313(d) authorizes EPA to add or delete chemicals from the list and sets criteria for these actions. EPCRA section 313(d)(2) states that EPA may add a chemical to the list if any of the listing criteria in Section 313(d)(2) are met. Therefore, to add a chemical, EPA must demonstrate that at least one criterion is met, but need not determine whether any other criterion is met. Conversely, to remove a chemical from the list, EPCRA section 313(d)(3) dictates that EPA must demonstrate that none of the listing criteria in Section 313(d)(2)(A) through (C) are met. The EPCRA section 313(d)(2)(A) through (C) criteria are: The chemical is known to cause or can reasonably be anticipated to cause significant adverse acute human health effects at concentration levels that are reasonably likely to exist beyond facility site boundaries as a result of continuous, or frequently recurring, releases. The chemical is known to cause or can reasonably be anticipated to cause in humans: [cir] Cancer or teratogenic effects; or [cir] Serious or irreversible-- [ssquf] Reproductive dysfunctions, [ssquf] Neurological disorders, [ssquf] Heritable genetic mutations; or [ssquf] Other chronic health effects. The chemical is known to cause or can be reasonably anticipated to cause, because of: [cir] Its toxicity; [cir] Its toxicity and persistence in the environment; or [cir] Its toxicity and tendency to bioaccumulate in the environment, a significant adverse effect on the environment of sufficient seriousness, in the judgment of the Administrator, to warrant reporting under this section. EPA often refers to the section 313(d)(2)(A) criterion as the ``acute human health effects criterion;'' the section 313(d)(2)(B) criterion as the ``chronic human health effects criterion;'' and the section 313(d)(2)(C) criterion as the ``environmental effects criterion.'' EPA published in the Federal Register of November 30, 1994 (59 FR 61432), a statement clarifying its interpretation of the section 313(d)(2) and (d)(3) criteria for modifying the section 313 list of toxic chemicals. III. Background Information A. What is the NTP and the report on carcinogens? The National Toxicology Program (NTP) is an interagency program within the Department of Health and Human Services (DHHS) headquartered at the National Institute of Environmental Health Sciences (NIEHS) of the National Institutes of Health (NIH). The mission of the NTP is to evaluate chemicals of public health concern by developing and applying tools of modern toxicology and molecular biology. The NTP program maintains an objective, science-based approach in dealing with critical issues in toxicology and is committed to using the best science available to prioritize, design, conduct, and interpret its studies. The mission of the NTP includes the evaluation of chemicals for their potential to cause cancer in humans. As part of their cancer evaluation work, the NTP periodically publishes a Report on Carcinogens (RoC) document. The RoC was mandated by the U.S. Congress, as part of the Public Health Service Act (Section 301(b)(4), as amended). The NTP describes the RoC as an informational scientific and public health document that identifies and discusses agents, substances, mixtures, or exposure circumstances that may pose a hazard to human health by virtue of their carcinogenicity. The NTP RoC serves as a meaningful and useful compilation of data on (1) the carcinogenicity (ability to cause cancer), genotoxicity (ability to damage genes), and biologic mechanisms (modes of action in the body) of the RoC-listed substances in humans and/or in animals, (2) the potential for human exposure to these substances, and (3) the regulations and guidelines promulgated by Federal agencies to limit exposures to RoC-listed substances. The NTP RoC is published periodically, with the most recently published 13th RoC having been released on October 2, 2014 (79 FR 60169, October 6, 2014). The 13th RoC contains the NTP cancer classifications from the most recent chemical evaluations, as well as the classifications from previous versions of the RoC (Reference (Ref.) 1). B. What are the NTP cancer classifications and criteria? The NTP RoC classifies chemicals as either ``known to be a human carcinogen'' or ``reasonably anticipated to be a human carcinogen.'' The criteria that the NTP uses to list an agent, substance, mixture, or exposure circumstance under each classification in the RoC (Ref. 2) are as follows: Known To Be Human Carcinogen: There is sufficient evidence of carcinogenicity from studies in humans *, which indicates a causal relationship between exposure to the agent, substance, or mixture, and human cancer. Reasonably Anticipated To Be Human Carcinogen: There is limited evidence of carcinogenicity from studies in humans *, which indicates that causal interpretation is credible, but that alternative explanations, such as chance, bias, or confounding factors, could not adequately be excluded, or there is sufficient evidence of carcinogenicity from studies in experimental animals, which indicates there is an increased incidence of malignant and/or a combination of malignant and benign tumors (1) in multiple species or at multiple tissue sites, or (2) by multiple routes of exposure, or (3) to an unusual degree with regard to incidence, site, or type of tumor, or age at onset, or there is less than sufficient evidence of carcinogenicity in humans or laboratory animals; however, the agent, substance, or mixture belongs to a well-defined, structurally related class of substances whose members are listed in a previous Report on Carcinogens as either known to be a human carcinogen or reasonably anticipated to be a human carcinogen, or there is convincing relevant information that the agent acts through mechanisms indicating it would likely cause cancer in humans. Conclusions regarding carcinogenicity in humans or experimental animals are based on scientific judgment, with consideration given to all relevant information. Relevant information includes, but is not limited to, dose response, route of exposure, chemical structure, metabolism, pharmacokinetics, sensitive sub-populations, genetic effects, or other data relating to mechanism of action or factors that may be unique to a given substance. For example, there may be substances for which there is evidence of carcinogenicity in laboratory animals, but there are compelling data indicating that the agent acts through mechanisms which do not operate in humans and would therefore not reasonably be anticipated to cause cancer in humans. * This evidence can include traditional cancer epidemiology studies, data from clinical studies, and/or data derived from the [[Page 20192]] study of tissues or cells from humans exposed to the substance in question, which can be useful for evaluating whether a relevant cancer mechanism is operating in humans. The NTP classifications for the potential for a chemical to cause cancer are very similar to the EPCRA section 313(d)(2)(B) statutory criteria for listing a chemical on the list of toxic chemicals subject to reporting under EPCRA section 313: ``(B) The chemical is known to cause or can reasonably be anticipated to cause in humans--(i) cancer . . .'' The specific data used by the NTP to classify a chemical as ``Known To Be Human Carcinogen'' or ``Reasonably Anticipated To Be Human Carcinogen'' are consistent with data used by EPA to evaluate chemicals for their potential to cause cancer and classify chemicals as either ``Carcinogenic to Humans'' or ``Likely to Be Carcinogenic to Humans'' (Ref. 3). C. What is the review process for the RoC? Specific details of the nomination and review process for the development of the 13th RoC are described in the Process for Preparation of the Report on Carcinogens section of the 13th RoC (Ref. 4). In general, the RoC review process includes evaluations by scientists from the NTP, other Federal health research and regulatory agencies (including EPA), and nongovernmental institutions. The RoC review process includes external peer review and several opportunities for public comment. For the 13th RoC, during the entire nomination, selection, and review process there were seven opportunities for public comment. For each candidate substance, an expert panel was convened to peer review the NTP monograph document prepared for each candidate substance. The RoC Monograph on 1-Bromopropane consists of the following components: (Part 1) the cancer evaluation component that reviews the relevant scientific information, assesses its quality, applies the RoC listing criteria to the scientific information, and gives the RoC listing status for 1-bromopropane, and (Part 2) the RoC monograph's substance profile containing the NTP's listing status decision, a summary of the scientific evidence considered key to reaching that decision, and data on properties, use, production, exposure, and Federal regulations and guidelines to reduce exposure to 1-bromopropane. The expert panel members had the following major responsibilities in reviewing the draft RoC monograph: (1) to comment on the draft cancer evaluation components for 1- bromopropane, specifically, whether they are technically correct and clearly stated, whether the NTP has objectively presented and assessed the scientific evidence, and whether the scientific evidence is adequate for applying the RoC listing criteria, and (2) to comment on the draft substance profile for 1-bromopropane, specifically, whether the scientific justification presented in the substance profile supports the NTP's preliminary policy decision on the RoC listing status of 1-bromopropane. The panel was also asked to vote on the following questions: (1) Whether the scientific evidence supports the NTP's conclusion on the level of evidence for carcinogenicity from experimental animal studies on 1-bromopropane and (2) whether the scientific evidence supports the NTP's preliminary listing decision for 1-bromopropane in the RoC. The panel agreed with the NTP conclusions that 1-bromopropane should be listed in the RoC based on sufficient evidence of carcinogenicity from studies in experimental animals, which found skin tumors in male rats, large intestine tumors in female and male rats, and lung tumors in female mice.'' (Ref. 5) Based upon the peer-review comments, the Office of the Report on Carcinogens (ORoC) prepared a revised draft RoC Monograph, which was then reviewed by the NTP Board of Scientific Counselors. The ORoC, in concert with the NTP Director, then finalized the RoC monographs and submitted the newly reviewed substances to the NTP Executive Committee for consultation. The final draft of the 13th RoC was then submitted to the Secretary of Health and Human Services (HHS) for review and approval. Once approved, the Secretary submitted the 13th RoC to the U.S. Congress as a final document and published the document on the RoC Web site (http://ntp.niehs.nih.gov/pubhealth/roc/roc13/index.html). IV. EPA's Review of the 13th RoC A. How did EPA select the NTP RoC chemical being proposed for addition? The most recent version of the NTP RoC that EPA previously reviewed for possible additions to the EPCRA section 313 list was the 12th RoC (March 13, 2013, 78 FR 15913). Each new version of the RoC adds newly classified chemicals to the existing list. EPA's present review of the 13th RoC identified 1-bromopropane as the only newly listed chemical that is not on the EPCRA section 313 list. EPA reviewed the NTP 13th RoC chemical profile and supporting materials for 1-bromopropane (Ref. 6). Given the extensive scientific reviews conducted by the NTP for their RoC documents, EPA's review focused on ensuring that there were no inconsistencies with how the Agency would consider the available data. EPA's review of the 1- bromopropane chemical profile and supporting material found no inconsistencies between how the data were interpreted by the NTP and how that same data would be interpreted under EPA's Guidelines for Carcinogen Risk Assessment (Ref. 3). Therefore, EPA agrees with the hazard conclusions of the NTP 13th RoC for 1-bromopropane. B. What technical data supports the NTP RoC classification and EPA's proposed addition of 1-bromopropane to the EPCRA section 313 list? This section presents the data that supported the NTP 13th RoC classification of 1-bromopropane and why EPA believes the data support the addition of this chemical to the EPCRA section 313 list. The RoC 1- Bromopropane Profile document (Ref. 7), the RoC Monograph on 1- Bromopropane (Ref. 5), and the available references cited within the portion of the 13th RoC chemical profile quoted here, are all included in the docket for this rulemaking. While they are contained in the docket and are part of the rulemaking record, the references within the quotation cited below from the 13th RoC 1-Bromopropane Profile document are not included in the list of references in Unit VI. of this Federal Register notice. The full citations for the references contained in the quotation can be found in the NTP 13th RoC 1-Bromopropane Profile document (Ref. 7). 1. 1-Bromopropane (Chemical Abstracts Service Registry Number 106- 94-5) (Refs. RoC Monograph and Profile documents (Refs. 5 and 7)). The NTP has classified 1-bromopropane as ``reasonably anticipated to be a human carcinogen.'' The classification is based on sufficient evidence of carcinogenicity in experimental animals and supporting data on mechanisms of carcinogenesis. The RoC substance profile for 1- bromopropane (Ref. 7) included the following summary information of the evidence of carcinogenicity: Carcinogenicity 1-Bromopropane is reasonably anticipated to be a human carcinogen based on sufficient evidence of carcinogenicity from studies in experimental animals. 1-Bromopropane, either directly or via reactive metabolites, causes molecular alterations that typically are associated with carcinogenesis, including genotoxicity, oxidative stress, and glutathione depletion. These alterations, observed mainly in vitro and in toxicity studies in rodents, are relevant to possible mechanisms of human carcinogenicity and support the relevance of the cancer studies in experimental animals to human carcinogenicity. [[Page 20193]] Cancer Studies in Experimental Animals Inhalation exposure to 1-bromopropane caused tumors in two rodent species and at several different tissue sites, including one tissue site in rats at which tumors are rare (NTP 2011). In male rats, 1-bromopropane caused significant dose-related increases in the incidences of several types of benign and/or malignant skin tumors (keratoacanthoma; keratoacanthoma and squamous-cell carcinoma combined; and keratoacanthoma, squamous-cell carcinoma, basal-cell adenoma, and basal-cell carcinoma combined). Both female and male rats showed an increased incidence of large- intestine tumors (adenoma of the colon and rectum), which are rare tumors in rats. In females, the incidence was dose-related and statistically significantly higher than in concurrent controls, and it exceeded the historical control range for all routes of exposure used in studies, including inhalation exposure. In males, the incidence of large-intestine adenoma was not significantly increased, but exceeded the historical control range for inhalation- exposure studies, and its occurrence was considered to be biologically significant because of the rarity of these tumors (which occurred in less than 0.2% of the historical controls). Although no carcinoma of the large intestine was observed in male or female rats in this study, adenoma of the large intestine has been shown to progress to carcinoma in other studies, and forms a morphologic continuum with carcinoma (Deschner 1983, Chang 1984, Nigro 1985). In female mice, 1-bromopropane caused significant dose-related increases in the incidence of benign and malignant lung tumors combined (alveolar/bronchiolar adenoma and carcinoma). These findings are supported by the observation of additional tumors in rats that may have been related to 1-bromopropane exposure, including malignant mesothelioma of the abdominal cavity and pancreatic islet tumors in males and skin tumors (squamous-cell papilloma, keratoacanthoma, and basal-cell adenoma or carcinoma) in females. Other Relevant Data 1-Bromopropane is well absorbed following ingestion, inhalation, or dermal exposure. Occupational exposure occurs primarily by inhalation and dermal contact. Unmetabolized 1-bromopropane has been detected in the urine of exposed workers at levels significantly correlated with exposure to 1-bromopropane in air (Kawai et al. 2001, Ichihara et al. 2004). 1-Bromopropane is metabolized via several pathways; 16 urinary metabolites have been detected in rodents, and several other metabolites have been proposed (Jones and Walsh 1979, Ishidao et al. 2002, Garner et al. 2006). The primary metabolic pathways in rodents are oxidation reactions catalyzed by cytochrome P450 (primarily CYP2E1) and glutathione conjugation. The available data on human metabolism of 1-bromopropane, although limited, suggest that some of its metabolic pathways in humans are similar to those observed in rodents. Four mercapturic conjugates identified in the urine of rodents were also identified in the urine of workers exposed to 1- bromopropane (Hanley et al. 2009). The major metabolite, N-acetyl-S- (n-propyl)-L-cysteine, has been detected in the urine of exposed workers at levels that increased with increasing levels of 1- bromopropane in ambient air (Hanley and Dunn 2006, Valentine et al. 2007, Hanley et al. 2009, 2010). This metabolite is produced in humans by conjugation of 1-bromopropane with glutathione, and that reaction also releases free bromide ions, another useful biomarker for human exposure to 1-bromopropane (Jones and Walsh 1979, Hanley et al. 2006). No studies were identified that tested for the occurrence in humans of the oxidative metabolites that are obligate intermediates to the measured conjugates. Studies on Mechanisms of Carcinogenesis The mechanism(s) by which 1-bromopropane causes cancer is not known. However, exposure to 1-bromopropane has been shown to cause molecular alterations related to carcinogenicity, including genotoxicity (mutations and DNA damage), oxidative stress, glutathione depletion, and immunomodulation. Studies have shown that 1-bromopropane can bind to macromolecules; it formed S-propylcysteine-globin adducts in exposed animals and humans (Valentine et al. 2007). Although 1-bromopropane did not induce mutations in bacteria under standard assay conditions, it did induce mutations in bacteria both with and without exogenous mammalian metabolic activation in the only reported study whose design was appropriate for testing a highly volatile chemical (Barber et al. 1981). It also caused mutations in cultured mammalian cells with or without mammalian metabolic activation (Elf Atochem 1996, as reviewed in NTP 2003) and DNA damage in cultured human cells without metabolic activation (Toraason et al. 2006). In addition, there is limited evidence of DNA damage in leukocytes from 1-bromopropane-exposed workers (Toraason et al. 2006). In rodents exposed in vivo, 1-bromopropane did not increase micronucleus formation in bone marrow (Kim et al. 1998, as reviewed in NTP 2003) or peripheral blood erythrocytes (Elf Atochem 1996, cited in NTP 2003, NTP 2011) or cause dominant lethal mutations. However, the dominant lethal mutation assay is generally regarded as relatively insensitive for the detection of mutagenic agents (Saito-Suzuki et al. 1982, Yu et al. 2008). There is evidence that metabolic activation plays a role in the genotoxicity and toxicity of 1-bromopropane. Several reactive metabolites (or intermediates) of 1-bromopropane have been identified in rodents, including glycidol and [alpha]-bromohydrin, and propylene oxide has been proposed as a metabolite (Garner et al. 2006). These compounds cause genotoxic effects in vitro, including DNA adduct formation, mutations, and DNA or chromosome damage (Stolzenberg and Hine 1979, IARC 1994, 2000). Glycidol and propylene oxide cause cytogenetic effects in vivo and are carcinogenic in experimental animals, and both substances are listed in the Report on Carcinogens as reasonably anticipated to be human carcinogens. These reactive and genotoxic metabolites may be responsible for at least some of the carcinogenic effects of 1-bromopropane. As with 1- bromopropane, oral exposure to glycidol caused rare tumors of the large intestine in rats, as did oral exposure to two halogenated alkane analogues of 1-bromopropane, tribromomethane and bromodichloromethane (NTP 1987, 1989, 1990). Chronic exposure to 1-bromopropane may produce levels of oxidative metabolites that exceed the glutathione-conjugating capacity or may inhibit enzymes required for glutathione synthesis. Because glutathione is an important cellular defense mechanism, reduced levels can lead to oxidative stress, increased toxicity, and carcinogenicity. Numerous studies have shown that 1-bromopropane induces both oxidative stress and glutathione depletion (Lee et al. 2005, 2007, 2010a, Liu et al. 2009, 2010, Huang et al. 2011). Studies with Cyp2e1-/- knockout mice, cytochrome P450 inhibitors, or a glutathione synthesis inhibitor showed that this metabolic activation pathway is involved in 1-bromopropane-induced toxicity, including neurological and reproductive effects, hepatotoxicity, and immunosuppression (NTP 2003, 2011, Lee et al. 2007, 2010a,b). Neurological effects of 1-bromopropane exposure have also been reported in humans (Li et al. 2010, Ichihara et al. 2012). It is unclear whether induction of immunotoxicity by 1- bromopropane plays a role in tumor development. Recent studies have shown that 1-bromopropane causes immunosuppression in rodents (Lee et al. 2007, Anderson et al. 2010). In particular, it reduced the numbers of T cells and T-cell subpopulations. In addition, there is evidence that 1-bromopropane causes an inflammatory response. It induced dose-related increases in gene expression and production of proinflammatory cytokines in mouse macrophages (Han et al. 2008) and an inflammatory response in rats (NTP 2011). However, chronic respiratory inflammation and lung tumors were not associated in rodents; respiratory inflammation occurred in rats but not mice, whereas lung tumors occurred in mice but not rats. Cancer Studies in Humans No epidemiological studies or case reports were identified that evaluated the relationship between human cancer and exposure specifically to 1-bromopropane. EPA has reviewed the NTP assessment for 1-bromopropane and agrees that 1-bromopropane can reasonably be anticipated to cause cancer in humans. EPA believes that the evidence is sufficient for listing 1- bromopropane on EPCRA section 313 pursuant to EPCRA section 313(d)(2)(B) based on the available carcinogenicity data for this chemical. V. Rationale for Listing The NTP RoC document undergoes significant scientific review and public [[Page 20194]] comment. The NTP review mirrors the review EPA has historically done to assess chemicals for listing under EPCRA section 313 on the basis of carcinogenicity. The conclusions regarding the potential for chemicals in the NTP RoC to cause cancer in humans are based on established sound scientific principles. EPA believes that the NTP RoC is an excellent and reliable source of information on the potential for chemicals covered in the NTP RoC to cause cancer in humans (see Unit III). Based on EPA's review of the data contained in the NTP 13th RoC, EPA has determined that 1-bromopropane can reasonably be anticipated to cause cancer (Ref. 6). Therefore, EPA believes that the evidence is sufficient for listing 1-bromopropane on the EPCRA section 313 toxic chemical list pursuant to EPCRA section 313(d)(2)(B) based on the available carcinogenicity data presented in the NTP 13th RoC. EPA considers chemicals that can reasonably be anticipated to cause cancer to have moderately high to high chronic toxicity. EPA does not believe that it is appropriate to consider exposure for chemicals that are moderately high to highly toxic based on a hazard assessment when determining if a chemical can be added for chronic effects pursuant to EPCRA section 313(d)(2)(B) (see 59 FR 61440-61442). Therefore, in accordance with EPA's standard policy on the use of exposure assessments (59 FR 61432), EPA does not believe that an exposure assessment is necessary or appropriate for determining whether 1- bromopropane meets the criteria of EPCRA section 313(d)(2)(B). VI. References EPA has established an official public docket for this action under Docket ID No. EPA-HQ-TRI-2015-0011. The public docket includes information considered by EPA in developing this action, including the documents listed below, which are electronically or physically located in the docket. In addition, interested parties should consult documents that are referenced in the documents that EPA has placed in the docket, regardless of whether these referenced documents are electronically or physically located in the docket. For assistance in locating documents that are referenced in documents that EPA has placed in the docket, but that are not electronically or physically located in the docket, please consult the person listed in the above FOR FURTHER INFORMATION CONTACT section. For convenience, the docket also includes all of the Federal Register documents cited in this action. 1. NTP, 2014. National Toxicology Program. Report on Carcinogens, Thirteenth Edition. Released October 2, 2014. U.S. Department of Health and Human Services, Public Health Service, National Toxicology Program, Research Triangle Park, NC 27709. (http://ntp.niehs.nih.gov/pubhealth/roc/roc13/index.html) 2. NTP, 2014. National Toxicology Program. Report on Carcinogens, Thirteenth Edition, Introduction section. Released October 2, 2014. U.S. Department of Health and Human Services, Public Health Service, National Toxicology Program, Research Triangle Park, NC 27709. 3. USEPA. Guidelines for Carcinogen Risk Assessment. Risk Assessment Forum, U.S. Environmental Protection Agency, Washington, DC, March 2005. 4. NTP, 2014. National Toxicology Program. Report on Carcinogens, Thirteenth Edition, Process for Preparation of the Report on Carcinogens section. Released October 2, 2014. U.S. Department of Health and Human Services, Public Health Service, National Toxicology Program, Research Triangle Park, NC 27709. 5. NTP, 2013. Report on Carcinogens Monograph on 1-Bromopropane. Office of the Report on Carcinogens, Division of the National Toxicology Program, National Institute of Environmental Health Sciences, U.S. Department of Health and Human Services. NIH Publication No. 13-5982, September 25, 2013 6. USEPA, OEI. Memorandum from Jocelyn Hospital, Toxicologist, Analytical Support Branch to Sandra Gaona, Acting Chief, Analytical Support Branch. November 3, 2014. Subject: Review of National Toxicology Program (NTP) Cancer Classification Data for 1- bromopropane. 7. NTP, 2014. National Toxicology Program. Report on Carcinogens, Thirteenth Edition, Profile for 1-Bromopropane. Released October 2, 2014. U.S. Department of Health and Human Services, Public Health Service, National Toxicology Program, Research Triangle Park, NC 27709. 8. USEPA, OEI. Economic Analysis of the Proposed Rule to add 1- Bromopropane to the EPCRA Section 313 List of Toxic Chemicals. February 17, 2015. VII. What are the Statutory and Executive Order reviews associated with this action? A. Executive Order 12866: Regulatory Planning and Review and Executive Order 13563: Improving Regulation and Regulatory Review This action is not a significant regulatory action and was therefore not submitted to the Office of Management and Budget (OMB) for review. B. Paperwork Reduction Act This action does not contain any new information collection requirements that require additional approval by the Office of Management and Budget (OMB) under the Paperwork Reduction Act (PRA). OMB has previously approved the information collection activities contained in the existing regulations and has assigned OMB control numbers 2025-0009 and 2050-0078. Currently, the facilities subject to the reporting requirements under EPCRA 313 and PPA 6607 may use either the EPA Toxic Chemicals Release Inventory Form R (EPA Form 1B9350-1), or the EPA Toxic Chemicals Release Inventory Form A (EPA Form 1B9350- 2). The Form R must be completed if a facility manufactures, processes, or otherwise uses any listed chemical above threshold quantities and meets certain other criteria. For the Form A, EPA established an alternative threshold for facilities with low annual reportable amounts of a listed toxic chemical. A facility that meets the appropriate reporting thresholds, but estimates that the total annual reportable amount of the chemical does not exceed 500 pounds per year, can take advantage of an alternative manufacture, process, or otherwise use threshold of 1 million pounds per year of the chemical, provided that certain conditions are met, and submit the Form A instead of the Form R. In addition, respondents may designate the specific chemical identity of a substance as a trade secret pursuant to EPCRA section 322, 42 U.S.C. 11042, 40 CFR part 350. OMB has approved the reporting and recordkeeping requirements related to Forms A and R, supplier notification, and petitions under OMB Control number 2025-0009 (EPA Information Collection Request (ICR) No. 1363) and those related to trade secret designations under OMB Control 2050-0078 (EPA ICR No. 1428). As provided in 5 CFR 1320.5(b) and 1320.6(a), an Agency may not conduct or sponsor, and a person is not required to respond to, a collection of information unless it displays a currently valid OMB control number. The OMB control numbers relevant to EPA's regulations are listed in 40 CFR part 9, 48 CFR chapter 15, and displayed on the information collection instruments (e.g., forms, instructions). C. Regulatory Flexibility Act (RFA), as Amended by the Small Business Regulatory Enforcement Fairness Act of 1996 (SBREFA), 5 U.S.C. 601 et seq. I certify that this action will not have a significant economic impact on a substantial number of small entities under the RFA. The Agency has determined that of the 140 entities estimated to be impacted by this action, 136 are small businesses; no small [[Page 20195]] governments or small organizations are expected to be affected by this action. All 136 small businesses affected by this action are estimated to incur annualized cost impacts of less than 1%. Facilities eligible to use Form A (those meeting the appropriate activity threshold which have 500 pounds per year or less of reportable amounts of the chemical) will have a lower burden. Thus, this action is not expected to have a significant adverse economic impact on a substantial number of small entities. A more detailed analysis of the impacts on small entities is located in EPA's economic analysis support document (Ref. 8). D. Unfunded Mandates Reform Act This action does not contain an unfunded mandate of $100 million or more as described in UMRA, 2 U.S.C. 1531 through 1538, and does not significantly or uniquely affect small governments. This action is not subject to the requirements of UMRA because it contains no regulatory requirements that might significantly or uniquely affect small governments. Small governments are not subject to the EPCRA section 313 reporting requirements. EPA's economic analysis indicates that the total cost of this action is estimated to be $531,002 in the first year of reporting (Ref. 8). E. Executive Order 13132 (Federalism) This action does not have federalism implications. It will not have substantial direct effects on the States, on the relationship between the national government and the States, or on the distribution of power and responsibilities among the various levels of government. F. Executive Order 13175: Consultation and Coordination With Indian Tribal Governments This action does not have tribal implications, as specified in Executive Order 13175. This action relates to toxic chemical reporting under EPCRA section 313, which primarily affects private sector facilities. Thus, Executive Order 13175 does not apply to this action. G. Executive Order 13045: Protection of Children From Environmental Health Risks and Safety Risks The EPA interprets Executive Order 13045 as applying only to those regulatory actions that concern environmental health or safety risks that the EPA has reason to believe may disproportionately affect children, per the definition of ``covered regulatory action'' in section 2-202 of the Executive Order. This action is not subject to Executive Order 13045 because it does not concern an environmental health risk or safety risk. H. Executive Order 13211: Actions Concerning Regulations That Significantly Affect Energy Supply, Distribution, or Use This action is not subject to Executive Order 13211, because it is not a significant regulatory action under Executive Order 12866. I. National Technology Transfer and Advancement Act This rulemaking does not involve technical standards. J. Executive Order 12898: Federal Actions To Address Environmental Justice in Minority Populations and Low-Income Populations The EPA believes the human health or environmental risk addressed by this action will not have potential disproportionately high and adverse human health or environmental effects on minority, low-income or indigenous populations. The results of this evaluation are contained below. This action does not address any human health or environmental risks and does not affect the level of protection provided to human health or the environment. This action adds an additional chemical to the EPCRA section 313 reporting requirements. By adding a chemical to the list of toxic chemicals subject to reporting under section 313 of EPCRA, EPA would be providing communities across the United States (including minority populations and low income populations) with access to data which they may use to seek lower exposures and consequently reductions in chemical risks for themselves and their children. This information can also be used by government agencies and others to identify potential problems, set priorities, and take appropriate steps to reduce any potential risks to human health and the environment. Therefore, the informational benefits of the action will have a positive impact on the human health and environmental impacts of minority populations, low-income populations, and children. List of Subjects in 40 CFR Part 372 Environmental protection, Community right-to-know, Reporting and recordkeeping requirements, and Toxic chemicals. Dated: April 8, 2015. Gina McCarthy, Administrator. Therefore, 40 CFR part 372 is proposed to be amended as follows: PART 372--TOXIC CHEMICAL RELEASE REPORTING: COMMUNITY RIGHT-TO-KNOW 0 1. The authority citation for part 372 continues to read as follows: Authority: 42 U.S.C. 11023 and 11048. 0 2. In Sec. 372.65, paragraph (a) is amended by adding in the table the entry for ``1-Bromopropane'' in alphabetical order and in paragraph (b) by adding in the table the entry for ``106-94-5'' in numerical order to read as follows: Sec. 372.65 Chemicals and chemical categories to which this part applies. * * * * * (a) * * * ------------------------------------------------------------------------ Effective Chemical name CAS No. date ------------------------------------------------------------------------ * * * * * 1-Bromopropane................................. 106-94-5 1/1/16 * * * * * ------------------------------------------------------------------------ (b) * * * ------------------------------------------------------------------------ Effective CAS No. Chemical name date ------------------------------------------------------------------------ * * * * * 106-94-5............................... 1-Bromopropane 1/1/16 * * * * * ------------------------------------------------------------------------ * * * * * [FR Doc. 2015-08664 Filed 4-14-15; 8:45 am] BILLING CODE 6560-50-P
![Federal Register / Vol. 80, No. 72 / Wednesday, April 15, 2015 / Proposed Rules 20189
b. How does a crew monitor and tower owner, turfing vendor, approaches had on work practices and
respond to changing weather subcontractor, and contractors hired by climber safety in those states?
conditions, including storms? the subcontractor all have some role in 36. Should an OSHA standard be
20. Fatigue: OSHA believes that the project) impacts employee safety at limited to work performed on
fatigue can affect communication tower communication tower worksites? communication towers, or should it also
workers in several ways. Climbing a 26. What practices might companies cover towers used for other purposes?
communication tower is physically in the contracting chain adopt to 37. If OSHA does not initiate a
demanding, and OSHA is concerned encourage communication and dedicated rulemaking for work on
that fatigue due to exertion can be coordination among employers at tower communication towers, what other
hazardous for tower workers. work sites? What obstacles stand in the types of regulatory actions might be
Accelerated work timelines can also way of communication and necessary and appropriate?
result in tower workers working very coordination between different parties 38. What non-regulatory approaches
long hours. And OSHA understands that in the contracting chain? could OSHA take to address hazards
communication tower workers may faced by employees working on
travel long distances to reach remote Economic Issues communication towers?
worksites, which can result in workers 27. The Agency seeks information on Authority and Signature
being fatigued before they even begin the number and size of firms that are
work. engaged in communication tower work This document was prepared under
a. What hazards are faced by a worker and on the number of employees the direction of David Michaels, Ph.D.,
who finds it physically challenging to employed by those firms. MPH, Assistant Secretary of Labor for
perform expected tasks, such as 28. The Agency seeks information Occupational Safety and Health, U.S.
climbing a tower or performing a self- about wage and turnover rates for Department of Labor. It is issued
rescue? What impact can this have on employees who work on pursuant to sections 3704 et seq., Public
other crew members? communication towers. The Agency is Law 107–217, 116 STAT. 1062 (40
b. What are the common causes of also interested in information about the U.S.C. 3704 et seq.); sections 4, 6, and
worker fatigue at communication tower experience possessed by workers 8, Public Law 91–596, 84 STAT. 1590
worksites? currently doing communication tower (29 U.S.C. 653, 655, 657); 29 CFR part
c. What are the effects of fatigue on work. Are they usually experienced in 1911; and Secretary of Labor’s Order No.
tower worker safety, and what types of this type of work? Are there many new 1–2012 (77 FR 3912 (Jan. 25, 2012)).
incidents occur as a result of worker or inexperienced employees working on Signed at Washington, DC, on March 27,
fatigue? communication towers? 2015.
21. Other common hazards: 29. What types of equipment are used David Michaels,
a. What other hazards are present in
in tower work and how often is this Assistant Secretary of Labor for Occupational
communication tower work, and what
equipment repaired and/or replaced? Safety and Health.
types of incidents are resulting from
30. The Agency seeks information [FR Doc. 2015–08633 Filed 4–14–15; 8:45 am]
those hazards? What can be done to
from all employers in the contracting BILLING CODE 4510–26–P
protect employees from those hazards?
b. What are some health and safety chain about the extent to which
considerations involved in working employees directly engaged in tower
with communications equipment work are covered by workers’ ENVIRONMENTAL PROTECTION
installed on non-dedicated tower compensation and/or an employer AGENCY
structures, such as water towers, liability insurance policy.
buildings, silos, electrical transmission 40 CFR Part 372
Tower Design
towers, etc.? [EPA–HQ–TRI–2015–0011; FRL–9925–29–
31. Can towers be designed and built OEI]
Contracting and Work Oversight with elevators for lifting personnel or
materials? Can towers be built with RIN 2025–AA41
22. Describe your role in the contract
chain and the key safety-related booms or davits aloft to aid in hoisting
materials? Addition of 1-Bromopropane;
provisions typically included in your Community Right-To-Know Toxic
contracts. How do contracting parties 32. How would elevators or davits
affect productivity/efficiency, e.g., the Chemical Release Reporting
oversee or enforce those provisions?
What are the consequences if a party amount of time spent on the tower? AGENCY: Environmental Protection
fails to fulfill those contractual How would elevators or davits address Agency (EPA).
requirements? or cause any safety hazards at the site? ACTION: Proposed rule.
23. What characteristics of past safety For example, would elevators or davits
performance does your company use in address hazards related to employee SUMMARY: The Environmental Protection
selecting potential contractors and fatigue? Agency (EPA) is proposing to add 1-
subcontractors? What safety-related 33. What are the industry standards bromopropane to the list of toxic
criteria does your company use in this for providing fall protection anchor chemicals subject to reporting under
selection process? points on new towers? section 313 of the Emergency Planning
24. Are safety-related factors and Community Right-to-Know Act
mstockstill on DSK4VPTVN1PROD with PROPOSALS
Regulatory/Non-Regulatory Approaches
considered in determining whether to (EPCRA) of 1986 and section 6607 of the
remove a contractor/subcontractor from 34. What would be the advantages Pollution Prevention Act (PPA) of 1990.
an ongoing project or from future and disadvantages of an OSHA standard 1-Bromopropane has been classified by
selection processes? If so, what specific that covers both construction and the National Toxicology Program in
factors are considered? maintenance activities on their 13th Report on Carcinogens as
25. What are the ways in which the communication towers? ‘‘reasonably anticipated to be a human
multi-leveled contracting environment 35. What effects have the North carcinogen.’’ EPA believes that 1-
(i.e., where entities such as the carrier, Carolina and Michigan regulatory bromopropane meets the EPCRA section
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![Federal Register / Vol. 80, No. 72 / Wednesday, April 15, 2015 / Proposed Rules 20195
governments or small organizations are subject to Executive Order 13045 Dated: April 8, 2015.
expected to be affected by this action. because it does not concern an Gina McCarthy,
All 136 small businesses affected by this environmental health risk or safety risk. Administrator.
action are estimated to incur annualized
cost impacts of less than 1%. Facilities H. Executive Order 13211: Actions Therefore, 40 CFR part 372 is
eligible to use Form A (those meeting Concerning Regulations That proposed to be amended as follows:
the appropriate activity threshold which Significantly Affect Energy Supply,
Distribution, or Use PART 372—TOXIC CHEMICAL
have 500 pounds per year or less of RELEASE REPORTING: COMMUNITY
reportable amounts of the chemical) will RIGHT-TO-KNOW
This action is not subject to Executive
have a lower burden. Thus, this action
Order 13211, because it is not a
is not expected to have a significant ■ 1. The authority citation for part 372
adverse economic impact on a significant regulatory action under
Executive Order 12866. continues to read as follows:
substantial number of small entities. A
Authority: 42 U.S.C. 11023 and 11048.
more detailed analysis of the impacts on I. National Technology Transfer and
small entities is located in EPA’s Advancement Act ■ 2. In § 372.65, paragraph (a) is
economic analysis support document amended by adding in the table the
(Ref. 8). This rulemaking does not involve entry for ‘‘1-Bromopropane’’ in
technical standards. alphabetical order and in paragraph (b)
D. Unfunded Mandates Reform Act by adding in the table the entry for
This action does not contain an J. Executive Order 12898: Federal ‘‘106–94–5’’ in numerical order to read
unfunded mandate of $100 million or Actions To Address Environmental as follows:
more as described in UMRA, 2 U.S.C. Justice in Minority Populations and
1531 through 1538, and does not Low-Income Populations § 372.65 Chemicals and chemical
significantly or uniquely affect small categories to which this part applies.
governments. This action is not subject The EPA believes the human health or * * * * *
to the requirements of UMRA because it environmental risk addressed by this (a) * * *
contains no regulatory requirements that action will not have potential
might significantly or uniquely affect disproportionately high and adverse Chemical name CAS No. Effective
human health or environmental effects date
small governments. Small governments
are not subject to the EPCRA section 313 on minority, low-income or indigenous
reporting requirements. EPA’s economic populations. The results of this * * * * *
analysis indicates that the total cost of evaluation are contained below. 1-Bromopropane ..... 106–94–5 1/1/16
this action is estimated to be $531,002 This action does not address any
in the first year of reporting (Ref. 8). * * * * *
human health or environmental risks
E. Executive Order 13132 (Federalism) and does not affect the level of
(b) * * *
protection provided to human health or
This action does not have federalism
implications. It will not have substantial the environment. This action adds an Effective
additional chemical to the EPCRA CAS No. Chemical name
direct effects on the States, on the date
relationship between the national section 313 reporting requirements. By
government and the States, or on the adding a chemical to the list of toxic
* * * * *
distribution of power and chemicals subject to reporting under
106–94–5 .. 1-Bromopropane 1/1/16
responsibilities among the various section 313 of EPCRA, EPA would be
levels of government. providing communities across the * * * * *
United States (including minority
F. Executive Order 13175: Consultation populations and low income * * * * *
and Coordination With Indian Tribal
populations) with access to data which [FR Doc. 2015–08664 Filed 4–14–15; 8:45 am]
Governments
they may use to seek lower exposures BILLING CODE 6560–50–P
This action does not have tribal and consequently reductions in
implications, as specified in Executive chemical risks for themselves and their
Order 13175. This action relates to toxic children. This information can also be FEDERAL COMMUNICATIONS
chemical reporting under EPCRA used by government agencies and others COMMISSION
section 313, which primarily affects to identify potential problems, set
private sector facilities. Thus, Executive priorities, and take appropriate steps to 47 CFR Part 73
Order 13175 does not apply to this
reduce any potential risks to human [MB Docket No. 15–88, RM–11747; DA 15–
action. 444]
health and the environment. Therefore,
G. Executive Order 13045: Protection of the informational benefits of the action
Children From Environmental Health will have a positive impact on the Television Broadcasting Services;
Risks and Safety Risks human health and environmental Bend, Oregon
The EPA interprets Executive Order impacts of minority populations, low- AGENCY: Federal Communications
mstockstill on DSK4VPTVN1PROD with PROPOSALS
13045 as applying only to those income populations, and children. Commission.
regulatory actions that concern ACTION: Proposed rule.
List of Subjects in 40 CFR Part 372
environmental health or safety risks that
the EPA has reason to believe may Environmental protection, SUMMARY: The Commission has before it
disproportionately affect children, per Community right-to-know, Reporting a petition for rulemaking filed by TDS
the definition of ‘‘covered regulatory and recordkeeping requirements, and Broadcasting LLC (‘‘TDS’’), the licensee
action’’ in section 2–202 of the Toxic chemicals. of KOHD, channel 51, Bend, Oregon,
Executive Order. This action is not requesting the substitution of channel
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Document Created: 2015-12-18 11:13:18
Document Modified: 2015-12-18 11:13:18
| Category | Regulatory Information | |
| Collection | Federal Register | |
| sudoc Class | AE 2.7: GS 4.107: AE 2.106: | |
| Publisher | Office of the Federal Register, National Archives and Records Administration | |
| Section | Proposed Rules | |
| Action | Proposed rule. | |
| Dates | Comments must be received on or before June 15, 2015. | |
| Contact | Daniel R. Bushman, Environmental Analysis Division, Office of Information Analysis and Access (2842T), Environmental Protection Agency, 1200 Pennsylvania Ave. NW., Washington, DC 20460; telephone number: 202-566-0743; fax number: 202- 566-0677; email: [email protected], for specific information on this notice. For general information on EPCRA section 313, contact the Emergency Planning and Community Right-to-Know Hotline, toll free at (800) 424-9346 (select menu option 3) or (703) 412-9810 in Virginia and Alaska or toll free, TDD (800) 553-7672, http://www.epa.gov/superfund/ contacts/infocenter/. | |
| FR Citation | 80 FR 20189 | |
| RIN Number | 2025-AA41 | |
| CFR Associated | Environmental Protection; Community Right-To-Know; Reporting and Recordkeeping Requirements and Toxic Chemicals |
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